Tasirin barasa na dogon lokaci akan kwakwalwa

Tasirin barasa na dogon lokaci akan kwakwalwa
Bayanai
Ƙaramin ɓangare na	long-term effects of alcohol consumption (en) , central nervous system disease (en) , alcohol-related brain damage (en) da nervous system alcohol-induced disorders (en)

Tasirin barasa na dogon lokaci akan kwakwalwa ya ƙunshi nau'ikan tasirin, bambanta ta hanyar sha, shekaru, kwayoyin halitta, da sauran abubuwan kiwon lafiya. Daga cikin gabobin da yawa da barasa ke shafar, kwakwalwa tana da rauni musamman. Yawan shan giya yana haifar da lalacewar kwakwalwar barasa, tare da barasa yana aiki a matsayin neurotoxin kai tsaye zuwa kwayoyin jijiyoyi, ^[1] yayin da ƙananan matakan shan barasa na iya haifar da raguwa a cikin ƙwayar kwakwalwa, ƙarar ƙwayar launin toka na yanki, da ƙananan kwayoyin halitta. Abincin barasa mai ƙarancin-zuwa matsakaici yana iya haɗawa da wasu fa'idodin fahimi ko neuroprotection a cikin tsofaffi. Abubuwan zamantakewa da tunani na iya ba da ƙananan tasirin kariya. Dangantaka gaba ɗaya tsakanin amfani da barasa da lafiyar kwakwalwa yana da rikitarwa, yana nuna ma'auni tsakanin tasirin neurotoxic barasa da tasirin daidaitawa.

Rabewa

Sakamakon jijiyoyi na amfani da barasa na dogon lokaci ana iya rarraba su cikin sakamako masu cutarwa (misali, atrophy na kwakwalwa, raguwar fahimi, lalata), yuwuwar tasirin tsaka tsaki, kuma, a iyakanceccen yanayi, ƙananan tasirin kariya masu alaƙa da ƙarancin-zuwa matsakaiciyar amfani. Ana rarraba bayyanar cututtuka masu tsanani a ƙarƙashin lalacewar kwakwalwar da ke da alaka da barasa (ARBD), ciki har da yanayi kamar ciwon Wernicke-Korsakoff da kuma ciwon barasa. ^[2]

Yawan shan barasa yana da alaƙa da tartsatsi da ƙananan raunuka na kwakwalwa, musamman lalacewa ga yankunan kwakwalwa ciki har da lobe na gaba, ^[3] tsarin limbic, da cerebellum, tare da atrophy na kwakwalwa mai yaduwa, ko raguwar kwakwalwa da lalacewa ta hanyar neuron . Ana iya ganin wannan lalacewa akan sikanin neuroimaging. ^[4] Barasa yana canza duka tsari da aikin kwakwalwa a sakamakon tasirin neurotoxic kai tsaye na barasa ko cirewar barasa. Ƙara yawan shan barasa yana haɗuwa da lalacewa ga yankunan kwakwalwa ciki har da lobe na gaba, tsarin limbic, da cerebellum, tare da yaduwa atrophy na cerebral, ko raguwar kwakwalwa da lalacewa ta hanyar neuron degeneration . Lalacewar lobe na gaba ya zama mafi shahara kamar shekarun barasa kuma yana iya haifar da ƙarancin aikin neuropsychological a cikin yankuna kamar warware matsala, kyakkyawan hukunci, da halayen jagoranci. ^[3] Rashin aikin sarrafa motsin rai yana haifar da lalacewa ga tsarin limbic. Wannan na iya haifar da matsala ga fahimtar yanayin fuska da kuma "fassara abubuwan motsin rai marasa fa'ida". ^[3]

Mummunan rikice-rikice sun haɗa da lalacewar kwakwalwa da ba za a iya jurewa ba, rashin lafiyar tabin hankali (misali, damuwa, damuwa), da ƙara haɗarin wasu yanayi na jijiyoyi, kamar cutar Alzheimer.

Manazarta

↑ "10th Special Report to the U.S. Congress on Alcohol and Health: Highlights from Current Research" (PDF). National Institute of Health. National Institute on Alcohol Abuse and Alcoholism. June 2000. p. 134. Archived from the original (PDF) on 21 February 2016. Retrieved 21 October 2014. The brain is a major target for the actions of alcohol, and heavy alcohol consumption has long been associated with brain damage. Studies clearly indicate that alcohol is neurotoxic, with direct effects on nerve cells. Chronic alcohol abusers are at additional risk for brain injury from related causes, such as poor nutrition, liver disease, and head trauma.
↑ Zahr, Natalie M.; Kaufman, Kimberley L.; Harper, Clive G. (May 2011). "Clinical and pathological features of alcohol-related brain damage". Nature Reviews Neurology (in Turanci). 7 (5): 284–294. doi:10.1038/nrneurol.2011.42. ISSN 1759-4758. PMC 8121189 Check |pmc= value (help). PMID 21487421.
1 2 3 Oscar-Berman, Marlene (June 2003). "Alcoholism and the Brain". Alcohol Research & Health. 27 (2): 125–133.
↑ Nutt, David; Hayes, Alexandra; Fonville, Leon; Zafar, Rayyan; Palmer, Emily O.C.; Paterson, Louise; Lingford-Hughes, Anne (2021-11-04). "Alcohol and the Brain". Nutrients (in Turanci). 13 (11): 3938. doi:10.3390/nu13113938. ISSN 2072-6643. PMC 8625009 Check |pmc= value (help). PMID 34836193 Check |pmid= value (help).

[1] "10th Special Report to the U.S. Congress on Alcohol and Health: Highlights from Current Research" (PDF). National Institute of Health. National Institute on Alcohol Abuse and Alcoholism. June 2000. p. 134. Archived from the original (PDF) on 21 February 2016. Retrieved 21 October 2014. The brain is a major target for the actions of alcohol, and heavy alcohol consumption has long been associated with brain damage. Studies clearly indicate that alcohol is neurotoxic, with direct effects on nerve cells. Chronic alcohol abusers are at additional risk for brain injury from related causes, such as poor nutrition, liver disease, and head trauma.

[2] Zahr, Natalie M.; Kaufman, Kimberley L.; Harper, Clive G. (May 2011). "Clinical and pathological features of alcohol-related brain damage". Nature Reviews Neurology (in Turanci). 7 (5): 284–294. doi:10.1038/nrneurol.2011.42. ISSN 1759-4758. PMC 8121189 Check |pmc= value (help). PMID 21487421.

[:0-3] 1 2 3 Oscar-Berman, Marlene (June 2003). "Alcoholism and the Brain". Alcohol Research & Health. 27 (2): 125–133.

[4] Nutt, David; Hayes, Alexandra; Fonville, Leon; Zafar, Rayyan; Palmer, Emily O.C.; Paterson, Louise; Lingford-Hughes, Anne (2021-11-04). "Alcohol and the Brain". Nutrients (in Turanci). 13 (11): 3938. doi:10.3390/nu13113938. ISSN 2072-6643. PMC 8625009 Check |pmc= value (help). PMID 34836193 Check |pmid= value (help).

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