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Cutar cututtuka

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Cutar cututtuka
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Cutar Septicemic tana ɗaya daga cikin nau'ikan annoba guda uku da Yersinia pestis ke haifarwa, wani nau'in ƙwayar cuta mai kama da gram-negative . Cutar Septicemic cuta ce ta tsarin jiki wadda Y. pestis ke kamuwa da jini cikin sauri, da farko tana haifar da alamun da ba su da takamaiman takamaiman, ciki har da zazzabi, sanyi, rashin lafiya, ciwon ciki, gudawa, da amai . Yayin da cutar ke ci gaba, toshewar jijiyoyin jini da ke yaɗuwa yana haifar da zubar jini na ciki da na waje, gangrene, da girgiza . Idan ba a yi magani ba, cutar septicemic kusan koyaushe tana kashe mutane, wani lokacin cikin awanni kaɗan bayan alamun farko. Ita ce ɗaya daga cikin nau'ikan annoba guda uku, tare da cutar bubonic (ta shafi ƙwayoyin lymph ) da kuma cutar huhu (ta shafi huhu ).

Ana yaɗa annobar cutar Septicemic ta hanyar ƙudaje masu kamuwa da cuta waɗanda ke rayuwa a kan ƙananan dabbobi masu shayarwa, amma kuma tana iya faruwa ne sakamakon mu'amala da dabbobi masu shayarwa da suka kamu da cutar. Tana iya tasowa kai tsaye daga kamuwa da cutar Y. pestis ko kuma a matsayin matsala ta annobar bubonic da ba a yi magani ba. A yanayin kamuwa da cutar ta farko, ƙwayoyin cuta na Y. pestis suna shiga fata bayan cizon ƙuda kuma suna yawo a cikin jinin jiki, ba tare da nuna alamun kamuwa da cuta ba. [1] Wannan yana sa ganewar asali ya zama da wahala, domin annoba gabaɗaya ba ta da yawa kuma alamun farko suna kama da wasu cututtukan jini da yawa da aka fi sani. Ana yin ganewar asali ta hanyar gano ƙwayoyin cuta a cikin jinin.[2][3][4][5][6][7][8][9][10]

Alamomi da Alamomi

[gyara sashe | gyara masomin]

Alamomin farko na annobar septicemic ba su da takamaiman takamaiman cututtuka, ciki har da zazzabi kwatsam, sanyi, da kuma rashin jin daɗi (rashin jin daɗi gaba ɗaya). Alamomin ciki, ciki har da ciwon ciki, gudawa, da amai, na iya zama, wanda hakan ke ƙara rikitar da ganewar farko. annobar septicemic na iya tasowa daga annobar bubonic da ba a yi magani ba, inda Y. pestis ke kamuwa da cuta kuma tana yaduwa a cikin ƙwayoyin lymph ; a cikin waɗannan yanayi, akwai alamun buboes . [11] Duk da haka, kamuwa da cutar jini ta farko ba ta bayyana tare da kowace buboes ba. [12] Waɗannan alamomin da ba su da takamaiman cututtuka suna sa ganewar asali ta yi wahala.

Yayin da ƙwayoyin cuta ke ci gaba da ƙaruwa a cikin jini, kamuwa da cuta yana haifar da toshewar jijiyoyin jini wanda ƙananan ɗigon jini ke samarwa a cikin jiki, yana amfani da abubuwan da ke haifar da ɗigon jini da platelets . Wannan na iya haifar da zubar jini a ƙarƙashin fata. Daga ƙarshe, kyallen takarda suna fara yin baƙi kuma suna mutuwa, suna haifar da gangrene na yatsu, yatsun ƙafa, hanci, da kunnuwa. Rashin ruwa a ƙarshe yana haifar da girgiza da gazawar gabobi . Idan kamuwa da cuta ya isa huhu, yanayin na iya ci gaba zuwa annobar huhu, [1] yana haifar da ciwon damuwa na numfashi mai tsanani (ARDS), wani nau'in gazawar numfashi mai kisa .

Cutar Septicemic plague na iya zama ba tare da wata alama ba kuma tana iya haifar da mutuwa ba tare da wata alama ba. [ <span title="This claim needs references to reliable sources. (June 2022)">ana buƙatar ambato</span> ]

Cututtukan Yersinia na ɗan adam galibi suna faruwa ne sakamakon cizon ƙuma da ta kamu da cutar ko kuma wani lokacin dabbar da ta kamu da cutar.

Beraye musamman dabbobi ne masu shayarwa waɗanda suka taka muhimmiyar rawa wajen yaɗuwar cutar. Idan beraye da ke ɗauke da cutar suka mutu sakamakon cutar, to ƙudajen da ke amfani da waɗannan beraye a matsayin tushen abinci dole ne su nemi wani jini daban da sauri, wanda hakan ke sa abubuwa su zama matsala ga duk wani ɗan adam da ke kusa.

Bayan ƙuma ta cinye jinin bera da ya kamu da cutar, ba za ta iya kai jini zuwa cikinta ba yayin da taruka ke samuwa saboda ƙwayoyin cuta na Yersinia da ke cikin ƙuma. Waɗannan taruka ba sa barin a sarrafa jini yadda ya kamata, suna barin ƙuma ta ji yunwa akai-akai, kuma wannan yana haifar da ƙarin cizo ga mutane. [13] Fuskantar cizon ƙuma daga ƙuma da ta kamu da cutar na iya haifar da annobar bubonic a cikin mutane wanda zai iya zama annobar septicemic.

Kuliyoyi da karnuka suma suna iya kamuwa da cizon ƙudaje masu kamuwa da cuta. Waɗannan kuliyoyi da karnuka za su iya fallasa mutane ga annobar lokacin da dabbar ta kawo waɗannan ƙudajen da suka kamu da cutar a kusa da mutane. [14]

Duk da haka, kamar yawancin cututtukan ƙwayoyin cuta na tsarin ƙwayoyin cuta, cutar na iya yaduwa ta hanyar buɗewa a cikin fata. Hakanan ana iya yada ta ta hanyar shaƙar digo na danshi daga atishawa ko tari, idan ƙwayoyin cuta suka bazu zuwa huhu kuma annobar huhu ta taso. A duka halayen biyu, annobar septicemic ba lallai ne ta zama sakamakon ba, musamman, ba sakamakon farko ba, amma lokaci-lokaci yana faruwa cewa annobar bubonic misali tana haifar da kamuwa da jini, kuma annobar septicemic ta haifar. Idan ƙwayoyin cuta suka shiga cikin jini maimakon lymph ko huhu, suna ninka a cikin jini, suna haifar da bacteremia da sepsis mai tsanani. A cikin annobar septicemic, endotoxins na ƙwayoyin cuta suna haifar da coagulation na jini da aka watsa (DIC), inda ƙananan ɗigon jini ke samuwa a ko'ina cikin jiki, wanda yawanci ke haifar da necrosis na ischemic, mutuwar nama saboda rashin zagayawa da kuma perfusion . [ ]

DIC yana haifar da raguwar albarkatun jini na jiki, ta yadda ba zai iya sake sarrafa zubar jini ba. Sakamakon haka, jinin da ba a zubar ba yana zubar jini a cikin fata da sauran gabobin jiki, wanda ke haifar da kuraje ja ko baƙi da kuma hematemesis (amai da jini) ko hemoptysis (tari da jini). Kurajen na iya haifar da kuraje a fata waɗanda suka yi kama da cizon kwari, yawanci ja, wani lokacin fari a tsakiya. [ ]

Ana kamuwa da annobar cutar Septicemic ta hanyar yaɗa cutar a kwance da kuma kai tsaye. Yaɗa cutar a kwance shine yaɗa cuta daga mutum ɗaya zuwa wani ba tare da la'akari da alaƙar jini ba. Yaɗa cutar kai tsaye yana faruwa ne ta hanyar hulɗa ta kusa da mutane, ta hanyar amfani da iska a kai a kai, ko kuma ta hanyar cizon ƙuma ko beraye da suka kamu da cutar. Yawancin beraye na iya ɗauke da ƙwayoyin cuta haka nan Leporidae kamar zomaye.

Kwayoyin cutar suna da alaƙa da duniya baki ɗaya, galibi a cikin beraye a duk nahiyoyi banda Ostiraliya da Antarctica. Mafi yawan kamuwa da cutar annoba ta ɗan adam yana faruwa ne a Afirka. Kwayoyin cutar sun fi bayyana a yankunan karkara da kuma duk inda akwai rashin tsafta, cunkoso, da yawan beraye a birane. Ayyukan waje kamar hawa dutse, sansani, ko farauta inda ake iya samun dabbobin da suka kamu da cutar, suna ƙara haɗarin kamuwa da cutar annoba, haka nan wasu ayyuka kamar na dabbobi ko wasu ayyukan da suka shafi dabbobi.

Ganewar Ganewa

[gyara sashe | gyara masomin]

Likita ko likitan dabbobi zai yi gwajin jiki wanda ya haɗa da tambaya game da tarihin lafiyar da kuma hanyoyin da za a iya kamuwa da cutar. Gwajin da zai iya haɗawa da:

  • Samfuran jini (gano ƙwayoyin rigakafi)
  • Samfuran ruwan jiki (duba ƙwayoyin cuta na Yersinia pestis )
  • Gwajin koda da hanta
  • Duba tsarin lymphatic don alamun kamuwa da cuta
  • Duba ruwan jiki don gano alamun da ba su dace ba
  • Dubawa don kumburi
  • Duba alamun rashin ruwa a jiki
  • Duba ko zazzaɓi ne
  • Duba kamuwa da cutar huhu

Ganewar bambance-bambance

[gyara sashe | gyara masomin]

Lamunin annoba na zamani ba kasafai ake samunsa ba, kuma alamun farko na annoba kamar mura da ta hanji ba su da takamaiman takamaiman bayani. Sakamakon haka, yawanci ba a zargin annobar har sai an tabbatar da kasancewar Y. pestis a jini. Lokacin da alamun cutar sepsis suka bayyana, ganewar asali ta haɗa da wasu cututtukan ƙwayoyin cuta da suka fi yawa, kamar cutar meningococcal da cutar endocarditis ta bakteriya . A cikin lamunin annoba inda akwai cutar matsananciyar numfashi (ARDS), ana iya zargin wasu yanayin numfashi kamar cutar hantavirus ko SARS . [1]

  1. 1 2 Kwit N, Nelson C, Kugeler K, Petersen J, Plante L, Yaglom H, Kramer V, Schwartz B, House J, Colton L, Feldpausch A, Drenzek C, Baumbach J, DiMenna M, Fisher E, Debess E, Buttke D, Weinburke M, Percy C, Schriefer M, Gage K, Mead P (28 August 2015). "Human Plague — United States, 2015". Morbidity and Mortality Weekly Report. 64 (33): 918–919. Cite error: Invalid <ref> tag; name "MMWR" defined multiple times with different content.
  2. Specchio N, Wirrell EC, Scheffer IE, Nabbout R, Riney K, Samia P, Guerreiro M, Gwer S, Zuberi SM, Wilmshurst JM, Yozawitz E, Pressler R, Hirsch E, Wiebe S, Cross HJ, Perucca E, Moshé SL, Tinuper P, Auvin S (June 2022). "International League Against Epilepsy classification and definition of epilepsy syndromes with onset in childhood: Position paper by the ILAE Task Force on Nosology and Definitions". Epilepsia. 63 (6): 1398–1442. doi:10.1111/epi.17241. PMID 35503717.
  3. Riney K, Bogacz A, Somerville E, Hirsch E, Nabbout R, Scheffer IE, Zuberi SM, Alsaadi T, Jain S, French J, Specchio N, Trinka E, Wiebe S, Auvin S, Cabral-Lim L, Naidoo A, Perucca E, Moshé SL, Wirrell EC, Tinuper P (June 2022). "International League Against Epilepsy classification and definition of epilepsy syndromes with onset at a variable age: position statement by the ILAE Task Force on Nosology and Definitions" (PDF). Epilepsia. 63 (6): 1443–74. doi:10.1111/epi.17240. PMID 35503725.
  4. Smith, RS; Kenny, CJ; Ganesh, V; Jang, A; Borges-Monroy, R; Partlow, JN; Hill, RS; Shin, T; Chen, AY; Doan, RN; Anttonen, AK; Ignatius, J; Medne, L; Bönnemann, CG; Hecht, JL; Salonen, O; Barkovich, AJ; Poduri, A; Wilke, M; de Wit, MCY; Mancini, GMS; Sztriha, L; Im, K; Amrom, D; Andermann, E; Paetau, R; Lehesjoki, AE; Walsh, CA; Lehtinen, MK (5 September 2018). "Sodium Channel SCN3A (NaV1.3) Regulation of Human Cerebral Cortical Folding and Oral Motor Development". Neuron. 99 (5): 905–913.e7. doi:10.1016/j.neuron.2018.07.052. PMC 6226006. PMID 30146301.
  5. Smith, Richard S.; Florio, Marta; Akula, Shyam K.; Neil, Jennifer E.; Wang, Yidi; Hill, R. Sean; Goldman, Melissa; Mullally, Christopher D.; Reed, Nora; Bello-Espinosa, Luis; Flores-Sarnat, Laura; Monteiro, Fabiola Paoli; Erasmo, Casella B.; Pinto e Vairo, Filippo; Morava, Eva; Barkovich, A. James; Gonzalez-Heydrich, Joseph; Brownstein, Catherine A.; McCarroll, Steven A.; Walsh, Christopher A. (22 June 2021). "Early role for a Na + ,K + -ATPase ( ATP1A3 ) in brain development". Proceedings of the National Academy of Sciences. 118 (25) e2023333118. doi:10.1073/pnas.2023333118. PMC 8237684. PMID 34161264
  6. Wu YW, Sullivan J, McDaniel SS, Meisler MH, Walsh EM, Li SX, Kuzniewicz MW (November 2015). "Incidence of Dravet Syndrome in a US Population". Pediatrics. 136 (5): e1310–5. doi:10.1542/peds.2015-1807. PMC 4621800. PMID 26438699.
  7. Cooper MS, Mcintosh A, Crompton DE, McMahon JM, Schneider A, Farrell K, Ganesan V, Gill D, Kivity S, Lerman-Sagie T, McLellan A, Pelekanos J, Ramesh V, Sadleir L, Wirrell E, Scheffer IE (December 2016). "Mortality in Dravet syndrome". Epilepsy Res. 128: 43–47. doi:10.1016/j.eplepsyres.2016.10.006. PMID 27810515.
  8. Bertrand D (2002). "How mutations in the nAChRs can cause ADNFLE epilepsy". Epilepsia. 43 Supple 5: 112–122. doi:10.1046/j.1528-1157.43.s.5.16.x. PMID 12121305.
  9. Loiseau P (1988). "Prognosis of benign childhood epilepsy with centro-temporal spikes. A follow-up of 168 patients". Epilepsia. 29 (3): 229–235. doi:10.1111/j.1528-1157.1988.tb03711.x. PMID 3371279. S2CID 13473409.
  10. Kuzniecky R, Rosenblatt B (1987). "Benign occipital epilepsy: a family study". Epilepsia. 24 (4): 346–350. doi:10.1111/j.1528-1157.1987.tb03655.x. PMID 3113923. S2CID 22014736.
  11. "Plague". MedlinePlus (in Turanci). United States National Library of Medicine. May 12, 2025. Retrieved June 22, 2026.
  12. Cite error: Invalid <ref> tag; no text was provided for refs named ":0".
  13. Zhou, Dongsheng; Han, Yanping; Yang, Ruifu (2006-01-01). "Molecular and physiological insights into plague transmission, virulence and etiology". Microbes and Infection. 8 (1): 273–284. doi:10.1016/j.micinf.2005.06.006. ISSN 1286-4579.
  14. CDC (2024-05-20). "How Plague Spreads". Plague (in Turanci). Retrieved 2024-09-25.