Gout

Daga Wikipedia, Insakulofidiya ta kyauta.

Gout wani nau'i ne na amosanin gabbai mai kumburi wanda ke da yawan hare-hare na ja, mai taushi, zafi, da kumburin haɗin gwiwa.[1][2] Ciwo yawanci yana zuwa da sauri, yana kaiwa ga mafi girman ƙarfi cikin ƙasa da sa'o'i 12.[3] Haɗin gwiwa a gindin babban yatsan yatsa yana shafar kusan rabin lokuta.[4] Hakanan yana iya haifar da tophi, duwatsun koda, ko lalacewar koda.[1]

Gout yana faruwa ne saboda yawan hawan uric acid a cikin jini.[2][3] Wannan yana faruwa ne daga haɗuwar abinci, wasu matsalolin lafiya, da abubuwan da ke haifar da kwayoyin halitta.[1][2] A babban matakan, uric acid crystallizes da lu'ulu'u ajiya ajiya a cikin gidajen abinci, tendons, da kewayen kyallen takarda, haifar da wani harin gout.[1] Gout yana faruwa a cikin waɗanda ke ci nama ko abincin teku akai-akai, suna shan giya, ko kuma suna da kiba.[1][5] Ana iya tabbatar da ganewar asali na gout ta kasancewar lu'ulu'u a cikin ruwan haɗin gwiwa ko a cikin ajiya a waje da haɗin gwiwa.[1] Matakan uric acid na jini na iya zama al'ada yayin hari.[1]

Jiyya tare da magungunan anti-inflammatory marasa steroidal (NSAIDs), steroids, ko colchicine suna inganta alamun bayyanar.[1][2][6] Da zarar mummunan harin ya ragu, ana iya rage matakan uric acid ta hanyar canje-canjen salon rayuwa kuma a cikin wadanda ke fama da hare-hare akai-akai, allopurinol ko probenecid suna ba da rigakafi na dogon lokaci.[3] Shan bitamin C da cin abinci mai yawa a cikin kayan kiwo maras kitse na iya zama rigakafi.[7][8]

Gout yana shafar kusan 1 zuwa 2% na al'ummar Yamma a wani lokaci a rayuwarsu.[3] Ya zama ruwan dare a cikin 'yan shekarun nan.[1] An yi imani da wannan saboda karuwar abubuwan haɗari a cikin yawan jama'a, irin su ciwo na rayuwa, tsawon rai, da canje-canje a cikin abinci.[3] Maza maza sun fi shafa.[1] A tarihi an san Gout da "cutar sarakuna" ko "cutar mai arziki".[3][9] An gane shi aƙalla tun daga zamanin Masarawa na da.[3]

Manazarta[gyara sashe | gyara masomin]

  1. 1.0 1.1 1.2 1.3 1.4 1.5 1.6 1.7 1.8 1.9 Dalbeth, N; Merriman, TR; Stamp, LK (April 2016). "Gout". Lancet (Review). 388 (10055): 2039–52. doi:10.1016/S0140-6736(16)00346-9. PMID 27112094.
  2. 2.0 2.1 2.2 2.3 Hui, M; Carr, A; Cameron, S; Davenport, G; Doherty, M; Forrester, H; Jenkins, W; Jordan, KM; Mallen, CD; McDonald, TM; Nuki, G; Pywell, A; Zhang, W; Roddy, E; British Society for Rheumatology Standards, Audit and Guidelines Working, Group. (26 May 2017). "The British Society for Rheumatology Guideline for the Management of Gout". Rheumatology (Oxford, England). 56 (7): e1–e20. doi:10.1093/rheumatology/kex156. PMID 28549177.
  3. 3.0 3.1 3.2 3.3 3.4 3.5 3.6 Richette P, Bardin T (January 2010). "Gout". Lancet. 375 (9711): 318–28. doi:10.1016/S0140-6736(09)60883-7. PMID 19692116.
  4. Schlesinger N (March 2010). "Diagnosing and treating gout: a review to aid primary care physicians". Postgrad Med. 122 (2): 157–61. doi:10.3810/pgm.2010.03.2133. PMID 20203467.
  5. Beyl Jr, R. N.; Hughes, L; Morgan, S (2016). "Update on Importance of Diet in Gout". The American Journal of Medicine. 129 (11): 1153–1158. doi:10.1016/j.amjmed.2016.06.040. PMID 27452679.
  6. Shekelle, P. G; Newberry, S. J; Fitzgerald, J. D; Motala, A; O'Hanlon, C. E; Tariq, A; Okunogbe, A; Han, D; Shanman, R (2017). "Management of Gout: A Systematic Review in Support of an American College of Physicians Clinical Practice Guideline". Annals of Internal Medicine. 166 (1): 37–51. doi:10.7326/M16-0461. PMID 27802478.
  7. "Questions and Answers about Gout". National Institute of Arthritis and Musculoskeletal and Skin Diseases. June 2015. Archived from the original on 15 January 2016. Retrieved 2 February 2016.
  8. Roddy, E; Choi, HK (May 2014). "Epidemiology of gout". Rheumatic Diseases Clinics of North America. 40 (2): 155–75. doi:10.1016/j.rdc.2014.01.001. PMC 4119792. PMID 24703341.
  9. "Rich Man's Disease – definition of Rich Man's Disease in the Medical dictionary". Free Online Medical Dictionary, Thesaurus and Encyclopedia.